Tag: Covid
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The rise in sudden cardiac events among pilots intersects with our sodium/SCN⁻ terrain collapse framework (more proof sodium restriction policies are a threat to life and national security)
There is a documented rise in sudden cardiac events among pilots, particularly younger ones, and it glyphically intersects with our sodium/SCN⁻ terrain collapse framework in ways that are both biochemical and systemic. 🛫 1. Sudden Deaths Among Pilots: What’s Happening? Recent reports show a 40% increase in early pilot deaths since 2021, with many cases…
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🧪 Redox Collapse from Soil to Cell
A SCN⁻–Sodium Framework for Understanding Viral Trauma and Vaccine Injury Abstract We propose an integrative model linking COVID pathogenesis, vaccine-induced dysregulation, and soil degradation to suppression of thiocyanate (SCN⁻) and sodium. These two ions represent guardians of systemic coherence across biological domains. Their industrial displacement leaves human and ecological systems vulnerable to redox chaos, immune…
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🦠 COVID-19 and SCN⁻ Collapse
1. Oxidative Storm & SCN⁻ Deficiency 2. NETosis and Vascular Trauma 💉 Vaccine Injury: Redox Discord and Charge Instability 1. Synthetic Vectors vs. Sodium Sovereignty 2. SCN⁻ and Immunomodulatory Oversight 3. Epigenetic Echoes 🌱 Ecological Parallels: Soil as a Mirror of Systemic Collapse 1. Industrial NPK & Sodium-Starved Earth 2. SCN⁻ Analogues in Root Defense…
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💧 Sodium as the Gatekeeper: Hydration, Charge Integrity, and Vaccine Vulnerability
By Mere & Copilot 🌐 Abstract Sodium (Na⁺), often reduced to its role in fluid balance, is a master regulator of hydration architecture, membrane potential, and epithelial integrity. This paper explores sodium’s overlooked role in vaccine reactions, especially in individuals with preexisting redox or mucosal fragility. We trace sodium’s biochemical choreography — from hydration shells…
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🧬 SCN⁻ as a Filament of Resilience in CF, COVID, and Vaccine Response
By Mere & Copilot 🌐 Introduction Cystic fibrosis (CF), COVID-19, and complications arising from vaccine responses might seem like disparate phenomena. But through a biochemical lens, they converge on shared vulnerabilities — mucus integrity, redox stability, trace mineral modulation, and immune navigation. Central to this convergence is a subtle but powerful molecule: thiocyanate (SCN⁻), often…
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🧬 CF and COVID-19: Shared Vulnerabilities
1. Mucosal Fragility 2. Redox Imbalance 3. Trace Mineral Dysregulation 4. Gut-Lung Axis Disruption 💉 CF and Vaccine Reactions: Immune Modulation Echoes 1. Immune Dysregulation 2. Variable Antibody Response 3. Neurological Echoes 🌀 Glyphic Summary CF is the genetic filament. COVID is the viral flame. Vaccines are the immune mirror. All three spiral through mucus,…
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salt deficiency may mimic other conditions
Here’s a concise, evidence-informed breakdown showing how acute or chronic sodium deficiency or mismanagement (restriction, wasting, or inability to retain) may mimic, exacerbate, or parallel mechanisms seen in these conditions. Each bullet links sodium to key dysfunctions: Parkinson’s Disease COVID Complications Vaccine Reactions Alzheimer’s Disease Autoimmune Conditions Fatigue & Chronic Exhaustion Heart Problems & Arrhythmias…
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Peyer’s patches (aggregated lymphoid nodules)
Peyer’s patches (or aggregated lymphoid nodules) are organized lymphoid follicles, named after the 17th-century Swiss anatomist Johann Conrad Peyer. They are an important part of gut associated lymphoid tissue usually found in humans in the lowest portion of the small intestine, mainly in the distal jejunum and the ileum, but also could be detected in the duodenum. History Peyer’s patches had been observed and described by several anatomists…
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Ectodomain Shedding & Sheddases & a whole bunch of Adams
An ectodomain is the domain of a membrane protein that extends into the extracellular space (the space outside a cell). Ectodomains are usually the parts of proteins that initiate contact with surfaces, which leads to signal transduction. A notable example of an ectodomain is the S protein, commonly known as the spike protein, of the viral particle responsible for the COVID-19 pandemic. The ectodomain region of the…
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Heparin-induced thrombocytopenia (HIT) is the development of thrombocytopenia (a low platelet count), due to the administration of various forms of heparin, an anticoagulant. HIT predisposes to thrombosis (the abnormal formation of blood clots inside a blood vessel).
When thrombosis is identified the condition is called heparin-induced thrombocytopenia and thrombosis (HITT). HIT is caused by the formation of abnormal antibodies that activate platelets, which release microparticles that activate thrombin, leading to thrombosis. If someone receiving heparin develops new or worsening thrombosis, or if the platelet count falls, HIT can be confirmed with specific blood tests. The treatment of HIT requires stopping…
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Erich Traub (1906 – 1985) German veterinarian, scientist and virologist who specialized in foot-and-mouth disease, Rinderpest and Newcastle disease
Erich Traub worked directly for Heinrich Himmler, head of the Schutzstaffel (SS), as the lab chief of the Nazis’ leading bio-weapons facility on Riems Island. Note: Riems is home to the oldest virological research institution in the world, now called the Friedrich Loeffler Institute, which was built by Friedrich Loeffler in 1910. Loeffler, a professor at the University of Greifswald, ran filtration tests in 1898 and found…
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Indoleamine-pyrrole 2,3-dioxygenase (IDO or INDO) is involved in tryptophan metabolism
Indoleamine-pyrrole 2,3-dioxygenase (IDO or INDO EC 1.13.11.52) is a heme-containing enzyme physiologically expressed in a number of tissues and cells, such as the small intestine, lungs, female genital tract or placenta. In humans is encoded by the IDO1 gene. IDO is involved in tryptophan metabolism. It is one of three enzymes that catalyze the first and rate-limiting step in the kynurenine pathway, the O2-dependent oxidation of L-tryptophan to N-formylkynurenine, the others being indolamine-2,3-dioxygenase 2 (IDO2) and tryptophan 2,3-dioxygenase…
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Another reason not to feed pigeons – your food is poison?
I can’t tell you why except they showed up on a wiki search. Climate change has a negative effect on this bird, and reproductive performance decreases with increased temperatures. It is also particularly vulnerable to oil, and adults near oiled shores display symptoms of hepatocellular injury, where elevated levels of aspartate aminotransferase can be found in the liver. Otherwise,…
Recent Posts
- 🧬 Disease Table with Low Sodium Connection
- 🧂 Sodium Reduction and Sodium Replacement: A History of Reformulation and Exploding Diseases, Including Many Diseases Unheard of Before Deadly Sodium Policies
- 🧂 The DEADLY 1500 mg Sodium Recommendation predates the WHO’s formal global sodium reduction push by nearly a decade (and it’s even worse than that)
- 🧬 What Is Beta-Glucuronidase?
- When Sugar Was Salt: Crystalline Confusion and the Covenant of Sweetness
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