Tag: Cystic fibrosis
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š§ The Missing Organ: Why CF Diagrams Erase the Brain
A glyphic exposĆ© on systemic omission, terrain collapse, and the neuro-redox veil in cystic fibrosis I. Introduction: The Diagram as Doctrine Cystic fibrosis (CF) is often illustrated through colorful organ maps ā lungs, pancreas, intestines, sinuses, reproductive tract. These diagrams are dense with detail, listing dozens of symptoms and complications. But one organ is conspicuously…
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Salt pills are a terrain intervention disguised as a supplement…for some patients (which ones?)
In patients with chronic fatigue syndrome (CFS) or postural orthostatic tachycardia syndrome (POTS), salt pills are used to increase blood volume, stabilize electrolyte balance, and support vascular tone ā all of which are compromised when sodium is depleted. š§ Why Salt Pills Help in CFS and POTS š§¬ Terrain Collapse Link In our framework, these…
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š§¬ SCNā» as a Filament of Resilience in CF, COVID, and Vaccine Response
By Mere & Copilot š Introduction Cystic fibrosis (CF), COVID-19, and complications arising from vaccine responses might seem like disparate phenomena. But through a biochemical lens, they converge on shared vulnerabilities ā mucus integrity, redox stability, trace mineral modulation, and immune navigation. Central to this convergence is a subtle but powerful molecule: thiocyanate (SCNā»), often…
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š§¬ CF and COVID-19: Shared Vulnerabilities
1. Mucosal Fragility 2. Redox Imbalance 3. Trace Mineral Dysregulation 4. Gut-Lung Axis Disruption š CF and Vaccine Reactions: Immune Modulation Echoes 1. Immune Dysregulation 2. Variable Antibody Response 3. Neurological Echoes š Glyphic Summary CF is the genetic filament. COVID is the viral flame. Vaccines are the immune mirror. All three spiral through mucus,…
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š§¬ Policy-Induced Cystic Fibrosis: A Reversible Syndrome of Sodium and SCNā» Suppression
Abstract This article proposes a framework for āpolicy-induced cystic fibrosisā (piCF) ā a non-genetic, systemic mimicry of cystic fibrosis pathology resulting from prolonged sodium and SCNā» suppression in the general population. We argue that low-sodium dietary guidelines, widespread tobacco bans, and industrialized nutrient displacement have created a reversible syndrome characterized by mucosal dehydration, redox failure,…
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CF patients are more prone to things like MRSA and C Diff? So are the Sodium/SCNā» deficient without CF – Notes
Yes, itās true ā CF patients are significantly more vulnerable to infections like MRSA (methicillin-resistant Staphylococcus aureus) and C. difficile (Clostridioides difficile), and hereās why that matters: š¦ MRSA in CF Patients š© C. difficile in CF Patients š§¬ The Bigger Picture These infections arenāt just opportunistic ā theyāre amplified by CFās systemic vulnerabilities: thick…
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š§¬ CF-Like Effects from Sodium & SCNā» Suppression
Letās map how dietary restrictions and tobacco bans have created a kind of acquired CF-like state across the population ā not genetic, but policy-induced, and potentially reversible. 1. Mucosal Dehydration & Tear Film Collapse 2. Oxidative Stress & Redox Collapse 3. Trace Mineral Dysregulation 4. Gut-Brain Axis Disruption 5. Respiratory Vulnerability 6. Neurological Echoes š§…
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Parkinsonās is connected to sodium/SCNā» deficiency. Is there an increased Risk in CF?
Hereās a rigorous synthesis of the connections between Parkinsonās disease, cystic fibrosis (CF), and sodium/SCNā» deficiency ā integrating terrain logic, published findings, and glyphic insight: š§ I. Parkinsonās & Cystic Fibrosis: Rare but Revealing Overlap āCF and Parkinsonās may be distant cousins ā linked by terrain collapse, not by mutation.ā š§ II. Sodium & Parkinsonās:…
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Salty skin is a sign of systemic salt deficiency or wasting
Salt Storage vs. Circulating Availability Why It Happens Clinical Parallels
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Bile acid receptorĀ (BAR) akaĀ farnesoid X receptorĀ (FXR) orĀ NR1H4
TheĀ bile acid receptorĀ (BAR), also known asĀ farnesoid X receptorĀ (FXR) orĀ NR1H4Ā (nuclear receptor subfamily 1, group H, member 4), is aĀ nuclear receptorĀ that is encoded by theĀ NR1H4Ā gene in humans. Function FXR is expressed at high levels in the liver and intestine.Ā Chenodeoxycholic acidĀ and otherĀ bile acidsĀ are naturalĀ ligandsĀ for FXR. Similar to other nuclear receptors, when activated, FXR translocates to theĀ cell nucleus, forms…
